์๋ ๋
ธํ ๋ถ๋ณ์ ๋ฒ์น ๊ธฐ์ฌ ๋ค๊ณ ์จ ๊ณ ๋์ด ๊ฒฐ๊ตญ ์ฐ๊ตฌ์ง๋ค ์ฐ๊ตฌ ํ ๋๋ก ์ํฐ์์ด์ง ์ฐ๊ตฌ๋ ํจ๊ณผ ์๋๊ฒ์ด๋ผ ํด์ ๋
ผ๋ฌธ ์์ฒด๊ฐ ๊ทธ๋ฐ์ง ์ง์ ์ฐพ์๋ดค์.
๊ฒฐ๋ก ๋ง ์ฐพ์์ ์ฝ์ด๋ดค๋๋ฐ ์ฌ๊ธฐ์ ๋ง์ง๋ง ๋๋ชฉ์ด ์ค์ํจ.
Finally, can we humans slow our own rate of ageing? Our findings support the idea that, in historical population when life expectancies were low, mortality improvements for infants, and in age-independent mortality, were the central contributors to the decades-long trend towards longer human life expectancies and greater lifespan equality3. These improvements were largely the result of environmental influences including social, economic, and public health advances13,34,35. Since the middle of the 20th century, however, declines in the baseline level of adult mortalityโmeasured in the context of the Siler model byย b0โhave very likely played an increasingly important role in industrialised societies3,8. As we show here, improvements in the environment are unlikely to translate into a substantial reduction in the rate of ageing,ย b1, or in the dramatic increase in lifespan that would result from such a change. It remains to be seen if future advances in medicine can overcome the biological constraints that we have identified here, and achieve what evolution has not.
๋
ผ๋ฌธ์์ ์ฌ๊ธฐ์ ์์๋ธ ์๋ฌผํ์ ์ ์ฝ์ ๊ทน๋ณตํ๊ณ ์ฐ๋ฆฌ ์์ ์ ๊ทผ๋ณธ์ ์ธ ๋
ธํ๋ฅผ ๋ฆ์ถ ์ ์๋์ง์ ๋ํด์๋ ๋ฏธ๋ ์ํ ๋ฐ์ ์ ๋ํด์ ๋๊ณ ๋ณผ ์ผ์ด๋ผ ๋์ด ์์. ๊ทธ๋ฆฌ๊ณ ์ด ๋
ผ๋ฌธ์์ ๋งํ๋ ์๋ฌผํ์ ์ ์ฝ์ ์ธ๊ฐ์ ๊ฒ๋ ๊ฐ์๊ฒ ์๋๋ผ ์ฌํ, ๊ฒฝ์ , ๊ณต์ค ๋ณด๊ฑด ๋ฑ, ๊ฒฐ๊ตญ์ ํ๊ฒฝ์ ์ธ ์ํฅ์ ์ด์นญํ๋๊ฒ์ผ๋ก ๋์ด ์์.
๊ฒฐ๋ก ์ ์ด ๋
ผ๋ฌธ์ ์ํฐ์์ด์ง ์ฐ๊ตฌ๊ฐ ๋ฌด์ธ๋ชจ๋ค ๋ผ๋ ๋ด์ฉ์ด๋์ ์์ ๊ด๋ จ์ด ์์. ๊ทธ๋ฆฌ๊ณ ์๋ช
๊ณผํ์ ๋ํ ์๊ธฐ์ง๋ง ์ฌํ๊ณผํ์ ์ธ ๋ถ๋ถ์ ๋ค๋ ํจ์ถํ๋ ๋
ผ๋ฌธ์.
- dc official App
์ ์ด์ Hypothesis๋๊น ๊ณผํ์ ํฉํธ๋ฅผ ๊ท๋ช ํ ๊ฒ๋ ์๋์ง
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